Exploring the effect of simulated Motion Conditions on Task Performance

This thesis explored the effect that four different areas of motion conditioning presented in a motion simulator had on defence-force based task performance. It is produced in conjunction with the Defence Science and Technology Organisation, Land Operations Division, to expand their understanding of how these conditions may affect their personnel. The four conditions are explored are Motion Sickness, Motion Fatigue, Motion Perception and Mental Workload under motion conditions. All studies involved first year psychology students enrolled at the University of Sydney in accordance with the University’s ethical guidelines (2013/388). In the study of Motion Sickness, nausea was shown to have very little detrimental effect on task performance. In long term driving exposure there was a slight negative effect on the reaction time that was linked to motion sickness symptoms. Two styles of driving were researched for Motion Fatigue: boredom and constant motion. The boredom drive was seen to have a slightly negative effect on performance compared to the motion drive. In analysing biomarkers of fatigue relevant to a defence context, the best indicators were respiratory rate and the Root Mean Square of Successive Differences between normal heart beats. In Motion Perception, 6-axes of motion at 3 intensities were tested using Defence force tasks to determine whether any one axis, or a certain intensity, negatively affected performance more than others. Higher errors occurred in the Roll direction. The Pitch direction was the least comfortable for participants. In the final chapter of Mental Workload under motion, increased workload did not have a great impact on performance, although further studies are needed. In an analysis of subjective scales of workload in simple tasks, participants were able to accurately determine their task performance. From a bio-measure perspective, pupil diameter and respiratory rate were found to be the most indicative of changing levels of workload

Australian Veterinary History Record No. 47

Australian Veterinary Associatio

Medulloblastomas and ependymal tumours; impact of translational research on treatment perspectives.

Contains fulltext : 50581.pdf (publisher's version ) (Open Access)RU Radboud Universiteit Nijmegen, 3 oktober 2006Promotor : Padberg, G.W.A.M. Co-promotores : Wesseling, P., Kappelle, A.C.VII, 134 p

Pathophysiological basis of contralateral reinnervation in facial palsy

Item does not contain fulltex

Demyelinating polyneuropathy in Leber hereditary optic neuropathy.

Contains fulltext : 51239.pdf (publisher's version ) (Closed access)We report a patient with Leber hereditary optic neuropathy (G11778A mtDNA) and a severe demyelinating neuropathy, for which no other cause except his mitochondrial disorder could be found. The involvement of the peripheral nervous system of patients with LHON, in particular with a 11778 mtDNA, is discussed

Malignant migrating partial seizures in a 4-month-old boy.

Item does not contain fulltex

Spontaneous intracranial hypotension in a patient with classical type Ehlers-Danlos syndrome.

Item does not contain fulltex

Recurrent varicella-zoster virus myelitis in an immunocompetent patient.

Item does not contain fulltex

[Not every TIA is primarily vascular]

Item does not contain fulltextThree patients, two women aged 72 and 45 years, and a man aged 80 years, presented with transient neurological deficits due to a brain tumour, a glioblastoma multiforme and two meningiomas respectively. A fourth patient, an 84-year-old man, had a transient ischaemic attack (TIA) with a meningioma as an incidental finding. The first woman had normal CT findings, but MRI revealed the neoplasm. Symptoms included motor loss, sensory disturbances, dysphasia and dysarthria, lasting from 30 seconds up to 10 minutes. The first two patients had surgery; the first one later died when the tumour recurred. The other two patients still exhibit a spontaneous recovery. Of all patients with a clinical presentation of a TIA, 0.4-1% harbour a brain tumour. Clinical symptoms do not distinguish 'transient tumour attacks' from TIAs with a primarily vascular origin. Transient tumour attacks are mainly seen with meningiomas, and to a lesser extent with high-grade gliomas. Changes in intracranial pressure leading to focal ischaemia may explain the occurrence of this phenomenon. A part from intracerebral tumours, non-vascular entities mimicking TIAs can also be seen with demyelinating processes, metabolic disturbances, epilepsy or migraine. Brain imaging is always required in patients with transient neurological deficits. A CT scan may provide false-negative results and in case of doubt, MRI is the preferred diagnostic tool